Genetic Obesity

 Genes can directly cause obesity disorders such as Bardet-Biedl syndrome and Prader-Willi syndrome. Obesity is a complex disease resulting from the interactions of a wide variety of hereditary and environmental factors. The advancement in quantitative genetics, genomics and bioinformatics has attributed to a better understanding of the genetic and molecular basis of obesity. Clustering the cases within a family, the congruence of body weight for Monozygotic twins, and the discovery of genes associated with obesity are key points that offering interest regarding the genetic dimension of obesity. Heritability of obesity vary depending on the phenotype studied, however it tends to be higher for phenotypes linked to adipose tissue distribution (40-55%) and for weight or body fat excess (5-40%). Weight gain and adiposity increase with age, an effect also influenced by heredity. Occurrences of monogenic types of obesity are evidence that it may be caused by Genetic Mutations, as only 78 cases worldwide have been attributed to mutations of seven distinct genes.

  • Epigenetics
  • Hereditary Factors
  • Clinical Genomics
  • Oncogenomics and Therapeutics
  • Nutrigenetics
  • Genome Wide Association Studies
  • Non-Syndromic Genetic Obesity

Related Conference of Genetic Obesity

July 17-18, 2017

11th Obesity and Endocrinology Congress

Melbourne, Australia
August 24-25, 2017

12th World Congress on Obesity

(10 Plenary Forums - 1Event) Toronto, Canada
September 21-22, 2017

13th Euro Obesity and Endocrinology Congress

Zurich, Switzerland
Oct 23-24, 2017

14th Global Obesity Meeting

Dubai, UAE
October 30- 31, 2017

15th International Conference on Obesity Medicine

Thailand, Bangkok

Genetic Obesity Conference Speakers

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